|
Ethicists
|
N05.350.262 |
|
|
2,2'-Dipyridyl
|
D03.383.725.220 |
|
|
Proline-Directed Protein Kinases
|
D08.811.913.696.620.682.700.646 |
|
|
T-Lymphocytes, Regulatory
|
A15.382.490.555.567.569.500.700 |
|
|
Micrognathism
|
C07.320.440.457 |
|
|
Disopyramide
|
D03.383.725.227 |
|
|
Brain Diseases, Metabolic
|
D001928 |
[Brain dysfunction or damage caused by acquired (i.e., non-inborn) metabolic disorders. Associated conditions include ENDOCRINE DISEASES; WATER-ELECTROLYTE IMBALANCE; KIDNEY DISEASES; LIVER DISEASES; anoxia (HYPOXIA, BRAIN); nutritional disorders (see NUTRITIONAL AND METABOLIC DISEASES); an encephalopathy associated with HEMODIALYSIS; and other disorders. (From Plum & Posner, Diagnosis of Stupor and Coma, 3rd ed, pp208-260)
, Acquired or inborn metabolic diseases that produce brain dysfunction or damage. These include primary (i.e., disorders intrinsic to the brain) and secondary (i.e., extracranial) metabolic conditions that adversely affect cerebral function.
] |
|
Brain Edema
|
D001929 |
[Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)
] |
|
Thioamides
|
D02.065.900 |
|
|
Keratitis, Herpetic
|
C01.375.725.465 |
|
|
Brain Death
|
D001926 |
[A state of prolonged irreversible cessation of all brain activity, including lower brain stem function with the complete absence of voluntary movements, responses to stimuli, brain stem reflexes, and spontaneous respirations. Reversible conditions which mimic this clinical state (e.g., sedative overdose, hypothermia, etc.) are excluded prior to making the determination of brain death. (From Adams et al., Principles of Neurology, 6th ed, pp348-9)
] |
|
Genes, sis
|
G05.360.340.024.340.375.500.791.560 |
|
|
Brain Diseases
|
D001927 |
[Pathologic conditions affecting the BRAIN, which is composed of the intracranial components of the CENTRAL NERVOUS SYSTEM. This includes (but is not limited to) the CEREBRAL CORTEX; intracranial white matter; BASAL GANGLIA; THALAMUS; HYPOTHALAMUS; BRAIN STEM; and CEREBELLUM.
] |
|
Autonomic Nerve Block
|
E04.525.210.550.500 |
|
|
Nerve Block
|
E04.525.210.550 |
|
|
Double Effect Principle
|
N05.350.238 |
|
|
Glomus Tympanicum
|
A08.713.228 |
|
|
Hemipelvectomy
|
E04.555.080.380 |
|
|
Clostridium botulinum type C
|
B03.353.625.375.500.160.150 |
|
|
Stargardt Disease
|
C16.320.290.724 |
|
