|
Peroxides
|
D02.389.338 |
|
|
Dental Restoration Failure
|
D019232 |
[Inability or inadequacy of a dental restoration or prosthesis to perform as expected.
] |
|
Retreatment
|
D019233 |
[The therapy of the same disease in a patient, with the same agent or procedure repeated after initial treatment, or with an additional or alternate measure or follow-up. It does not include therapy which requires more than one administration of a therapeutic agent or regimen. Retreatment is often used with reference to a different modality when the original one was inadequate, harmful, or unsuccessful.
] |
|
Dentinal Fluid
|
D019234 |
[The lymph or fluid of dentin. It is a transudate of extracellular fluid, mainly cytoplasm of odontoblastic processes, from the dental pulp via the dentinal tubules. It is also called dental lymph. (From Stedman, 26th ed, p665)
] |
|
Noonan Syndrome
|
C05.660.207.690 |
|
|
Dental Audit
|
D019230 |
[A detailed review and evaluation of selected clinical records by qualified professional personnel for evaluating quality of dental care.
] |
|
Subarachnoid Hemorrhage, Traumatic
|
D020206 |
[Bleeding into the SUBARACHNOID SPACE due to CRANIOCEREBRAL TRAUMA. Minor hemorrhages may be asymptomatic; moderate to severe hemorrhages may be associated with INTRACRANIAL HYPERTENSION and VASOSPASM, INTRACRANIAL.
] |
|
Tracheobronchomalacia
|
C16.131.621.953 |
|
|
Coma, Post-Head Injury
|
D020207 |
[Prolonged unconsciousness from which the individual cannot be aroused, associated with traumatic injuries to the BRAIN. This may be defined as unconsciousness persisting for 6 hours or longer. Coma results from injury to both cerebral hemispheres or the RETICULAR FORMATION of the BRAIN STEM. Contributing mechanisms include DIFFUSE AXONAL INJURY and BRAIN EDEMA. (From J Neurotrauma 1997 Oct;14(10):699-713)
] |
|
Wolfram Syndrome
|
C16.320.290.564.980 |
|
|
Optic Atrophies, Hereditary
|
C16.320.290.564 |
|
|
Karnofsky Performance Status
|
E05.318.308.980.438.475.456.500.500 |
|
|
Acetoxyacetylaminofluorene
|
D02.241.081.018.110.080.070 |
|
|
Lasers, Solid-State
|
E07.710.520.490 |
|
|
Skull Fracture, Depressed
|
D020204 |
[A skull fracture characterized by inward depression of a fragment or section of cranial bone, often compressing the underlying dura mater and brain. Depressed cranial fractures which feature open skin wounds that communicate with skull fragments are referred to as compound depressed skull fractures.
] |
|
Insulin-Like Growth Factor II
|
D12.776.467.937.420 |
|
|
Skull Fracture, Basilar
|
D020205 |
[Fractures which extend through the base of the SKULL, usually involving the PETROUS BONE. Battle's sign (characterized by skin discoloration due to extravasation of blood into the subcutaneous tissue behind the ear and over the mastoid process), CRANIAL NEUROPATHIES, TRAUMATIC; CAROTID-CAVERNOUS SINUS FISTULA; and CEREBROSPINAL FLUID OTORRHEA are relatively frequent sequelae of this condition. (Adams et al., Principles of Neurology, 6th ed, p876)
] |
|
Adenosine Monophosphate
|
D13.695.827.068.180 |
|
|
Cerebral Hemorrhage, Traumatic
|
D020202 |
[Bleeding into one or both CEREBRAL HEMISPHERES due to TRAUMA. Hemorrhage may involve any part of the CEREBRAL CORTEX and the BASAL GANGLIA. Depending on the severity of bleeding, clinical features may include SEIZURES; APHASIA; VISION DISORDERS; MOVEMENT DISORDERS; PARALYSIS; and COMA.
] |
|
Brain Stem Hemorrhage, Traumatic
|
D020203 |
[Bleeding into structures of BRAIN STEM, including the MIDBRAIN; PONS; or MEDULLA OBLONGATA, as the result of CRANIOCEREBRAL TRAUMA. DIFFUSE AXONAL INJURY is commonly associated. Clinical manifestations may include OCULAR MOTILITY DISORDERS; ATAXIA; PARALYSIS; PERSISTENT VEGETATIVE STATE; and COMA.
] |
