|
Lagenidium
|
D044726 |
[A genus of OOMYCETES in the family Lagenidiaceae. One species (L. giganteum) parasitizes the larval stage of MOSQUITOES and is used in its biological control (PEST CONTROL, BIOLOGICAL); it also causes disease in freshwater FISHES.
] |
|
Pancrelipase
|
D020799 |
[A preparation of hog pancreatic enzymes standardized for lipase content.
] |
|
Facial Nerve Diseases
|
D005155 |
[Diseases of the facial nerve or nuclei. Pontine disorders may affect the facial nuclei or nerve fascicle. The nerve may be involved intracranially, along its course through the petrous portion of the temporal bone, or along its extracranial course. Clinical manifestations include facial muscle weakness, loss of taste from the anterior tongue, hyperacusis, and decreased lacrimation.
] |
|
Receptor, Platelet-Derived Growth Factor alpha
|
D020796 |
[A PDGF receptor that binds specifically to both PDGF-A chains and PDGF-B chains. It contains a protein-tyrosine kinase activity that is involved in SIGNAL TRANSDUCTION.
] |
|
Facial Neuralgia
|
D005156 |
[Neuralgic syndromes which feature chronic or recurrent FACIAL PAIN as the primary manifestation of disease. Disorders of the trigeminal and facial nerves are frequently associated with these conditions.
] |
|
Ileal Neoplasms
|
C06.405.469.420.501 |
|
|
Receptor, Platelet-Derived Growth Factor beta
|
D020797 |
[A PDGF receptor that binds specifically to the PDGF-B chain. It contains a protein-tyrosine kinase activity that is involved in SIGNAL TRANSDUCTION.
] |
|
Facial Pain
|
D005157 |
[Pain in the facial region including orofacial pain and craniofacial pain. Associated conditions include local inflammatory and neoplastic disorders and neuralgic syndromes involving the trigeminal, facial, and glossopharyngeal nerves. Conditions which feature recurrent or persistent facial pain as the primary manifestation of disease are referred to as FACIAL PAIN SYNDROMES.
] |
|
Prunus
|
B01.650.940.800.575.912.250.859.937.500.625 |
|
|
Receptor Protein-Tyrosine Kinases
|
D020794 |
[A class of cellular receptors that have an intrinsic PROTEIN-TYROSINE KINASE activity.
] |
|
Laurencia
|
D044722 |
[A genus of RED ALGAE in the family Rhodomelaceae. Some species are a rich source of chlorine-and bromine-containing metabolites which show significant antibacterial activity.
] |
|
Facial Paralysis
|
D005158 |
[Severe or complete loss of facial muscle motor function. This condition may result from central or peripheral lesions. Damage to CNS motor pathways from the cerebral cortex to the facial nuclei in the pons leads to facial weakness that generally spares the forehead muscles. FACIAL NERVE DISEASES generally results in generalized hemifacial weakness. NEUROMUSCULAR JUNCTION DISEASES and MUSCULAR DISEASES may also cause facial paralysis or paresis.
] |
|
Photophobia
|
D020795 |
[Abnormal sensitivity to light. This may occur as a manifestation of EYE DISEASES; MIGRAINE; SUBARACHNOID HEMORRHAGE; MENINGITIS; and other disorders. Photophobia may also occur in association with DEPRESSION and other MENTAL DISORDERS.
] |
|
Factitious Disorders
|
D005162 |
[Disorders characterized by physical or psychological symptoms that are not real, genuine, or natural.
] |
|
Factor Analysis, Statistical
|
D005163 |
[A set of statistical methods for analyzing the correlations among several variables in order to estimate the number of fundamental dimensions that underlie the observed data and to describe and measure those dimensions. It is used frequently in the development of scoring systems for rating scales and questionnaires.
] |
|
Living Wills
|
I01.880.604.583.020.500 |
|
|
Advance Directives
|
I01.880.604.583.020 |
|
|
Factor IX
|
D005164 |
[Storage-stable blood coagulation factor acting in the intrinsic pathway of blood coagulation. Its activated form, IXa, forms a complex with factor VIII and calcium on platelet factor 3 to activate factor X to Xa. Deficiency of factor IX results in HEMOPHILIA B (Christmas Disease).
] |
|
Factor V
|
D005165 |
[Heat- and storage-labile plasma glycoprotein which accelerates the conversion of prothrombin to thrombin in blood coagulation. Factor V accomplishes this by forming a complex with factor Xa, phospholipid, and calcium (prothrombinase complex). Deficiency of factor V leads to Owren's disease.
] |
|
Curriculum
|
I02.158 |
|
